InterviewSolution
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InterviewSolution
This section includes InterviewSolutions, each offering curated multiple-choice questions to sharpen your knowledge and support exam preparation. Choose a topic below to get started.
| 51. |
Tricorrhexis nodosa is a characteristic finding of (A) Argininosuccinic aciduria (B) Citrullinemia (C) Phenylketonuria (D) Hyperargininemia |
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Answer» (D) Hyperargininemia |
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| 52. |
If glucose-1-phosphate formed by glycogenoloysis in muscles is oxidized to CO2 and H2O, the energy yield will be (A) 2 ATP equivalents (B) 3 ATP equivalents (C) 4 ATP equivalents (D) 8 ATP equivalents |
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Answer» (B) 3 ATP equivalents |
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| 53. |
Adenylate cyclase is activated by (A) Insulin (B) Glucagon (C) Prostaglandin E1 (D) Ca2+ ions |
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Answer» (B) Glucagon |
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| 54. |
The output of free fatty acids from adipose tissue is reduced by (A) Insulin (B) Glucagon (C) Growth hormone (D) Epinephrine |
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Answer» The output of free fatty acids from adipose tissue is reduced by Insulin. |
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| 55. |
The desaturation and chain elongation system of polyunsaturated fatty acid are enhanced by (A) Insulin (B) Glucagon (C) Epinephrine (D) Thyroxine |
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Answer» The desaturation and chain elongation system of polyunsaturated fatty acid are enhanced by Insulin. |
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| 56. |
Absence of the enzyme argininosuccinate synthetase causes (A) Argininosuccinic aciduria (B) Hyperargininemia (C) Tricorrhexis nodosa (D) Citrullinemia |
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Answer» (A) Argininosuccinic aciduria |
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| 57. |
A specific fructokinase present in liver has a very high affinity for its substrate because (A) Km for fructose is very high (B) Km for fructose is very low (C) Activity is affected by fasting (D) Activity is affected by insulin |
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Answer» (B) Km for fructose is very low |
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| 58. |
Hepatic glycogenoloysis is increased by (A) Insulin (B) Glucagon (C) Epinephrine (D) Glucocorticoids |
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Answer» (B) Glucagon |
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| 59. |
Glycogen phosphorylase liberates the following from glycogen (A) Glucose (B) Glucose-6-phosphate (C) Glucose-1-phosphate (D) Maltose |
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Answer» (C) Glucose-1-phosphate |
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| 60. |
Glycogen synthetase catalyses the formation of (A) α−1, 4-Glycosidic bonds (B) α−1, 6-Glycosidic bonds (C) Both (A) and (B) (D) None of these |
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Answer» (A) α−1, 4-Glycosidic bonds |
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| 61. |
Insulin has no effect on the activity of the enzyme: (A) Glycogen synthetase (B) Fructokinase (C) Pyruvate kinase (D) Pyruvate dehydrogenase |
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Answer» (B) Fructokinase |
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| 62. |
The glycogen content of the liver is upto (A) 6% (B) 8% (C) 10% (D) 12% |
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Answer» The glycogen content of the liver is upto 6%. |
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| 63. |
An example of hydrogen transferring coenzyme is (A) CoA (B) NAD+ (C) Biotin (D) TPP |
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Answer» An example of hydrogen transferring coenzyme is NAD+. |
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| 64. |
The main pathway for denovo synthesis of fatty acids occur in (A) Cytosol (B) Mitochondria (C) Microsomes (D) Nucleus |
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Answer» Chain elongation of fatty acids in mammalian liver occurs in Cytosol. |
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| 65. |
Acute hemolytic episode after administration of antimalarial, primaquin, is due to deficiency of the enzyme: (A) 6-Phosphogluconate dehydrogenase (B) Glucose-6-phosphate dehydrogenase (C) Epimerase (D) Transketolase |
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Answer» (B) Glucose-6-phosphate dehydrogenase |
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| 66. |
Acute hemolytic anemia in person’s sensitive to the Fava beans is due to the deficiency of the enzyme: (A) Pyruvate dehydrogenase (B) G-6-PD (C) Aconitase (D) Transketolase |
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Answer» The Correct option is G-6-PD. |
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| 67. |
The coenzyme not involved in the formation of acetyl-CoA from pyruvate is (A) TPP (B) Biotin (C) NAD (D) FAD |
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Answer» The coenzyme not involved in the formation of acetyl-CoA from pyruvate is Biotin. |
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| 68. |
Correct ordering of lipoprotein molecules from lowest to the greater density is (A) LDL, IDL, VLDL, chylomicron (B) Chylomicron, VLDL, IDL, LDL (C) VLDL, IDL, LDL, chylomicron (D) LDL, VLDL, IDL, chylomicron |
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Answer» (B) Chylomicron, VLDL, IDL, LDL |
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| 69. |
‘Clearing factor’ is (A) Lipoprotein lipase (B) Crotonase (C) 7-dehydro cholesterol (D) β-sitosterol |
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Answer» (A) Lipoprotein lipase |
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| 70. |
The major source of extracellular cholesterol for human tissue is (A) Very low density lipoprotein (B) High density lipoprotein (C) Low density lipoprotein (D) Albumin |
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Answer» (C) Low density lipoprotein |
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| 71. |
In humans under normal conditions loss of ketone bodies via urine is usually less than (A) 1 mg/24 hr (B) 4 mg/24 hr (C) 8 mg/24 hr (D) 10 mg/24 hr |
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Answer» (A) 1 mg/24 hr |
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| 72. |
The active site of an enzyme is formed by a few of the enzymes: (A) R groups of the amino acids (B) Amino groups of the amino acids (C) Carboxyl group of the amino acids (D) Exposed sulfur bonds |
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Answer» (C) Carboxyl group of the amino acids |
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| 73. |
Alkaloids are usually purified by extraction with (A) Ether (B) Dil HCl (C) NaOH (D) Chloroform |
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Answer» Alkaloids are usually purified by extraction with Dil HCl. |
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| 74. |
The number of N-MC groups in alkaloids is best estimate with the help of (A) HI (B) H2SO4 (C) (CH3CO)2 CO (D) CH3 Mg I |
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Answer» The Correct option is (A) HI |
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| 75. |
The affected organ in Mc Ardle’s syndrome is (A) Liver (B) Kidney (C) Liver and Heart (D) Skeletal muscle |
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Answer» (D) Skeletal muscle |
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| 76. |
Mc Ardle’s syndrome is characterized by the absence of (A) Liver phosphorylase (B) Muscle phosphorylase (C) Branching enzyme (D) Debranching enzyme |
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Answer» Mc Ardle’s syndrome is characterized by the absence of Muscle phosphorylase. |
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| 77. |
Tarui disease is characterized by the deficiency of the enzyme: (A) Liver phosphorylase (B) Muscle phosphorylase (C) Muscle and erythrocyte phosphofructokinase (D) Lysosomal acid maltase |
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Answer» (C) Muscle and erythrocyte phosphofructokinase |
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| 78. |
A specific inhibitor for succinate dehydrogenase is (A) Arsenite (B) Malonate (C) Citrate (D) Fluoride |
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Answer» (B) Malonate |
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| 79. |
A specific inhibitor for succinate dehydrogenase is (A) Arsenine (B) Arsenite (C) Citrate (D) Fluoride |
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Answer» A specific inhibitor for succinate dehydrogenase is Arsenite. |
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| 80. |
The most useful test for the diagnosis of acute hemorrhagic pancreatitis during the first few days is (A) Urinary lipase test (B) Serum calcium (C) Urinary amylase (D) Serum amylase |
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Answer» (D) Serum amylase |
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| 81. |
From arachidonate, synthesis of prostaglandins is catalysed by(A) Cyclooxygenase(B) Lipoxygenase(C) Thromboxane synthase(D) Isomerase |
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Answer» (A) Cyclooxygenase |
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| 82. |
In glycogenesis a branch point in the molecule is established by the enzyme (A) Amylo[1→ 4][1→ 6] transglucosidase (B) α [1→ 4] α [1→ 4] Glucan transferase (C) Amylo [1→ 6] glucosidase (D) Glycogen synthase |
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Answer» (A) Amylo[1→ 4][1→ 6] transglucosidase |
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| 83. |
In glycogenolysis, the enzyme which transfers a trisaccharide unit from one branch to the other exposing 1→ 6 branch point is (A) Phosphorylase (B) α-[1→ 4]→ α-[1→ 4]→ Glucan transferase (C) Amylo [1→ 6] glucosidase (D) Amylo[1→ 4]→ [1→ 6] transglucosidase |
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Answer» (B) α-[1→ 4]→ α-[1→ 4]→ Glucan transferase |
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| 84. |
Glucose-1-phosphate liberated from glycogen cannot be converted into free glucose in (A) Liver (B) Kidneys (C) Muscles (D) Brain |
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Answer» The Correct option is (C) Muscles |
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| 85. |
All the following are coenzymes except (A) Ubiquinone (B) CoA (C) Pyruvate dehydrogenase (D) Lipoic acid |
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Answer» (C) Pyruvate dehydrogenase |
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| 86. |
A flavoprotein in pyruvate dehydrogenase complex is (A) Pyruvate dehydrogenase (B) Didrolipoyl acetyl transferase (C) Dihydrolipoyl dehydrogenase (D) None of these |
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Answer» (C) Dihydrolipoyl dehydrogenase |
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| 87. |
An allosteric inhibitor of pyruvate dehydrogenase is (A) Acetyl CoA (B) ATP (C) NADH (D) Pyruvate |
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Answer» (A) Acetyl CoA |
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| 88. |
Bassen-Kornzweig syndrome is due to (A) Absence of Apo-C-II (B) Defect in Apo-B synthesis (C) Absence of Apo-E (D) Absence of Apo-D |
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Answer» (B) Defect in Apo-B synthesis |
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| 89. |
Hyperargininemia, a defect in urea synthesis develops due to deficiency of the enzyme: (A) Ornithine transcarbamoylase (B) Argininosuccinase (C) Arginase (D) Argininosuccinate synthetase |
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Answer» (B) Argininosuccinase |
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| 90. |
Dehydrogenases utilize, as coenzymes, all of the following except (A) NAD+ (B) NADP+ (C) FAD (D) FH4 |
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Answer» Dehydrogenases utilize, as coenzymes, all of the following except FH4. |
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| 91. |
Urease is a (A) Lyase (B) Ligase (C) Isomerase (D) Hydrolase |
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Answer» Urease is a Hydrolase. |
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| 92. |
Urea is produced physiologically by the action of the enzyme: (A) Urease (B) Glutaminase (C) Arginase (D) None of these |
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Answer» (C) Arginase |
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| 93. |
The kinetic effect of purely competitive inhibitor of an enzyme (A) Increases Km without affecting Vmax (B) Decreases Km without affecting Vmax (C) Increases Vmax without affecting Km (D) Decreases Vmax without affecting Km |
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Answer» (A) Increases Km without affecting Vmax |
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| 94. |
Fatty liver is also caused by (A) CH3Cl (B) CCl4 (C) Na2SO4 (D) Riboflavin |
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Answer» Fatty liver is also caused by CCl4. |
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| 95. |
Immobilized enzymes: (A) Potentiation of activity (B) Presentation of activity (C) Preparation of activity (D) All of these |
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Answer» Correct option (B) Presentation of activity Explanation: Certain enzymes can be made to bind to insoluble inorganic matrix (e.g., cyanogens bromide activated sepharose) to preserve their catalytic activity for long periods. Such enzymes are referred to as immobilized enzymes. |
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| 96. |
The hormone activating the glycogen synthase activity is (A) Insulin (B) Glucagon (C) Epinephrine (D) ACTH |
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Answer» The hormone activating the glycogen synthase activity is Insulin. |
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| 97. |
Action of glycogen synthase is inhibited by (A) Insulin (B) Glucose (C) Mg2+ (D) Cyclic AMP |
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Answer» Action of glycogen synthase is inhibited by Mg2+. |
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| 98. |
The fatty acid synthase complex catalyses (A) 4 sequential enzymatic steps (B) 6 sequential enzymatic steps (C) 7 sequential enzymatic steps (D) 8 sequential enzymatic steps |
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Answer» (B) 6 sequential enzymatic steps |
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| 99. |
IUB had divided enzymes into how many classes? (A) 6 (B) 5 (C) 8 (D) 4 |
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Answer» IUB had divided enzymes into 6 classes. |
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| 100. |
The induced fit model of enzyme action was proposed by (A) Fischer (B) Koshland (C) Mitchell (D) Markert |
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Answer» (B) Koshland |
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